diff --git a/content/3.defense-systems/abid.md b/content/3.defense-systems/abid.md
index f41523d23aa0cb0364af67c5c369ee186252b381..a74aaebac0340b5e46f8e1b5ba92b80cce0930d1 100644
--- a/content/3.defense-systems/abid.md
+++ b/content/3.defense-systems/abid.md
@@ -10,10 +10,28 @@ tableColumns:
     Activator: Unknown
     Effector: Unknown
     PFAM: PF07751
+contributors:
+  - Marian Dominguez-Mirazo
+  - Florian Tesson
+relevantAbstracts:
+    - doi: 10.1023/A:1002027321171
+    - doi: 10.1016/j.mib.2005.06.006
+    - doi: 10.1128/aem.61.5.2023-2026.1995
 ---
 
 # AbiD
 
+## Description
+
+AbiD is a single gene system system discovered in may 1995 in the plasmid pBF61 of *Lactococcus lactis* :ref{doi=10.1128/aem.61.5.2023-2026.1995}. An homologs of AbiD, named AbiD1 was discovered in july 1995 :ref{doi=10.1128/jb.177.13.3818-3823.1995}.
+
+AbiD is one of the so-called "Abi" systems for "Abortive infection" discovered in the 90's in research related to the dairy industry :ref{doi=10.1016/j.mib.2005.06.006}. AbiR is classified as possible abortive infection in :ref{doi=10.1016/j.mib.2023.102312}.
+
+This antiphage defense system is very close to AbiD1 and AbiF :ref{doi=10.1128/aem.61.12.4321-4328.1995} and are categorized as the same system. Furtheremore, AbiD is also detected as the [Abi2](/defense-systems/abi2) system.
+### Molecular mechanism
+
+For AbiD1 :ref{doi=10.1128/jb.177.13.3824-3829.1995} ([AAA79209](https://www.ncbi.nlm.nih.gov/protein/AAA79209)), the expression of the protein is toxic to the bacteria and is therefore highly regulated. Expression of the protein is triggered by the orf1 of phage bIL66 which results in inhibition of the phage RuvC-like endonuclease activity and stops phage multiplication.
+
 ## Example of genomic structure
 
 The AbiD is composed of 1 protein: AbiD.
@@ -72,14 +90,3 @@ end
     style Title3 fill:none,stroke:none,stroke-width:none
     style Title4 fill:none,stroke:none,stroke-width:none
 </mermaid>
-## Relevant abstracts
-
-::relevant-abstracts
----
-items:
-    - doi: 10.1023/A:1002027321171
-    - doi: 10.1016/j.mib.2005.06.006
-
----
-::
-