From 93625c79ec285ee386780356fd5ee0098c03a342 Mon Sep 17 00:00:00 2001
From: ftesson <florian.tesson@pasteur.fr>
Date: Fri, 19 Jan 2024 15:07:36 +0100
Subject: [PATCH] merge main correct misspelling

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 content/3.defense-systems/abid.md | 7 ++++---
 1 file changed, 4 insertions(+), 3 deletions(-)

diff --git a/content/3.defense-systems/abid.md b/content/3.defense-systems/abid.md
index c383291c..2844485c 100644
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+++ b/content/3.defense-systems/abid.md
@@ -23,11 +23,12 @@ relevantAbstracts:
 
 ## Description
 
-AbiD is a single gene system system discovered in may 1995 in the plasmid pBF61 of *Lactococcus lactis* :ref{doi=10.1128/aem.61.5.2023-2026.1995}. An homologs of AbiD, named AbiD1 was discovered in july 1995 :ref{doi=10.1128/jb.177.13.3818-3823.1995}.
+AbiD is a single gene system system discovered in May 1995 in the plasmid pBF61 of *Lactococcus lactis* :ref{doi=10.1128/aem.61.5.2023-2026.1995}. An homolog of AbiD, named AbiD1 was discovered in July 1995 :ref{doi=10.1128/jb.177.13.3818-3823.1995}.
 
-AbiD is one of the so-called "Abi" systems for "Abortive infection" discovered in the 90's in research related to the dairy industry :ref{doi=10.1016/j.mib.2005.06.006}. AbiR is classified as possible abortive infection in :ref{doi=10.1016/j.mib.2023.102312}.
+AbiD is one of the so-called "Abi" systems for "Abortive infection" discovered in the 90's in research related to the dairy industry :ref{doi=10.1016/j.mib.2005.06.006}. AbiR is classified as a possible abortive infection in :ref{doi=10.1016/j.mib.2023.102312}.
+
+This antiphage defense system is very close to AbiD1 and AbiF :ref{doi=10.1128/aem.61.12.4321-4328.1995} and is categorized as the same system. Furthermore, AbiD is also detected as the [Abi2](/defense-systems/abi2) system.
 
-This antiphage defense system is very close to AbiD1 and AbiF :ref{doi=10.1128/aem.61.12.4321-4328.1995} and are categorized as the same system. Furtheremore, AbiD is also detected as the [Abi2](/defense-systems/abi2) system.
 ### Molecular mechanism
 
 For AbiD1 :ref{doi=10.1128/jb.177.13.3824-3829.1995} ([AAA79209](https://www.ncbi.nlm.nih.gov/protein/AAA79209)), the expression of the protein is toxic to the bacteria and is therefore highly regulated. Expression of the protein is triggered by the orf1 of phage bIL66 which results in inhibition of the phage RuvC-like endonuclease activity and stops phage multiplication.
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